参考文献
[1]Potkin BN, Barorelli AL, Gessert JM, et al. Coronary Artery Imaging with Intravascular High-frequency Ultrasound. Circulation,1990; 81: 1575-1585.
[2]Tobis JM, Mallery J, Mahon D, et al. Intravascular Ultrasound Imaging of Human Coronary Arteries in Vivo. Circulation, 1991;83: 913-926.
[3]Mintz GS, Nissen SE, Anderson WD, et al. American College of Cardiology Clinical Expert Consensus Document on Standards for Acquisition, Measurement and Reporting of Intravascular Ultrasound Studies (IVUS): A Report of the American College of Cardiology Task Force on Clinical Expert Consensus Documents. J Am Coll Cardiol, 2001; 37: 1478-1492.
[4]Peters RJ, Kow WE, Havenith MG, et al. Histopathologic Validation of Intravascular Ultrasound Imaging. J Am Soc Echocardiogr,1994; 7: 230-241.
[5]Kimura BJ, Bhargava, and DeMaria AN. Value and Limitations of Intravascular Ultrasound Imaging in Characterizing Coronary Atherosclerotic Plaque. Am Heart J, 1995; 130: 386-396.
[6]Nair A, Kuban BD, Tuzcu EM, et al. Coronary Plaque Classification With Intravascular Ultrasound Radiofrequency Data Analysis. Circulationm, 2002; 106: 2200-2206.
[7]Moore MP, Spencer T, Salter DM, et al. Characterization of Coronary Atherosclerotic Morphology by Spectral Analysis of Radiofrequency Signal: In Vitro Intravascular Ultrasound Study with Histological and Radiological Validation. Heart, 1998; 79:459-467.
[8]Watson RJ, MeLean CC, Moore MP, et al. Characterization of Arterial Plaque by Spectral Analysis of In Vivo Radiofrequency Intravascular Ultrasound Data. Ultrasound Med Biol, 2000; 26: 73-80.
[9]Nasu K, Tsuchikane E, Katoh O, et al. Accuracy of In Vivo Coronary Plaque Morphology Assessment: A Validation Study of In Vivo Virtual Histology Compared With In Vitro Histopathology. J Am Coll Cardiol, 2006; 47: 2405-2412.
[10]Nair A, Margolis MP, Kuban BD . et al. Automated Coronary Plaque Characterization With Intravascular Ultrasound Backscatter: Ex Vivo Validation. Eurointerv, 2007; 3: 113-120.
[11]Virmani R, Kolodgie FD, Burke AP, et al. Lessons Learn Sudden Coronary Death: A Comprehensive Morphological Classification Scheme for Atherosclerotic Lesions. Arterioscler Throm Vasc Biol, 2000; 20: 1261-1275.
[12]Virmani R, Burke AP, Farb A . et al. Pathology of the Vulnerable Plaque. J Am Coll Cardial, 2006; 47: C13-C18.
[13]Burke AP, Farb A, Malcom GT, et al. Plaque Rupture and Sudden Death Related to Exertion in Men with Coronary Artery Disease. JAMA, 1999; 281: 921-926.
[14]Burke AP, Farb A, Malcom GT, et al. Effect of Risk Factors on the Mechanism of Acute Thrombosis and Sudden Coronary Death in Women. Circulation, 1998; 97: 210-2116.
[15]Giroud D, Li JM, Urban P, et al. Relation of the Site of Acute Myocardial Infarction to the Most Severe Coronary Arterial Stenosis at Prior Angiography. Am J Cardiol, 1992; 69: 729-732.
[16]Kolodgie FD, Burke AP, Farb A, et al. The Thin-cap Fibroatheroma Type of Vulnerable Plaque: The Major Precursor Lesion to Acute Coronary Syndromes. Curr Opin Cardiol, 2001; 16: 285-292.
[17]Virmani R, Burke AP, Kolodgie FD, et al. Pathology of the Thin-Cap Fibroatheroma: A Type of Vulnerable Plaque. J. Interv Cardiol, 2003; 16: 267-272.
[18]Rodriguez-Granillo GA, Garcia-Garcia HM, McFadden E, et al. In Vivo Intravascular Ultrasound-Derived Thin-Cap Fibroatheroma Detection sing Ultrasound Radiofrequency Data Analysis. . J Am Coll Cardial, 2005; 46: 2038-2042.
[19]Hong MK, Mintz GS, Lee CW, et al. A Three Vessel Virtual Histology Intravascular Ultrasound Analysis of Frequency and Distribution of Thin-Cap Fibroatheromas in Patients With Acute Coronary Syndrome or Stable Angina Pectoris. Am J Cardiol,2008; 101: 568-572.
[20]Wang JC, Normad SL, Mauri L, et al. Coronary Artery Spatial Distribution of Acute Myocardial Infarction Occlusions.Circulation, 2004; 110: 278-284.
[21]Vagimigli M, Rodriguez-Granillo GA, Garcia-Garcia HM, et al. Distance From the Ostium as an Independent Determinant of Coronary Plaque Composition In Vivo: an Intravascular Ultrasound Study Based Radiofrequency Data Analysis in Humans. Eur Heart J, 2006; 26: 655-663.
[22]Nakamura M, Nishikawa H, Mukai S, et al. Impact of Coronary Artery Remodeling on Clinical Presentation of Coronary Artery Disease; An Intravascular Ultrasound Study. J Am Coll Cardial, 2001; 37: 63-69.
[23]Rodriguez-Granillo GA, Serruys PW, Garcia-Garcia HM, et al. Coronary Artery Remodeling is Related to Plaque Composition.Heart, 2006; 92: 388-391.
[24]Surmely JF, Nasu K, Fujita H, et al. Association of Coronary Plaque Composition and Arterial Remodeling: A Virtual Histology Intravascular Ultrasound Analysis. Heart, 2007; 93: 928-932.
[25]Stone GW, Maehara A, Lansky AJ, et al. A Prospective Natural History Study of Coronary Atherosclerosis. N Engl J Med, 2011;364: 226-235.
[26]Calvert PA, Obaid DR, O’Sullivan M, et al. Assoication Between IVUS Findings and Adverse Outcomes in Patients With Coronary Artery Disease. J Am Coll Cardiol Img, 2011; 4: 894-901.
[27]Cheng JM, Garcia-Carcia HM, de Boer SP, et al. In Vivo Detection of High-risk Coronary Plaques by Radiofrequency Intravascular Ultrasound and Cardiovascular Outcome: Results of the ATHEROREMO-IVUS Study. Eur Heart J, 2014; 35:639-647.
[28]Pian RN, Paik GY, Moscucci M, et al. Incidence and Treatment of “No-refolw” After Percutaneous Coronary Intervention.Circulation, 1994; 89: 2514-2518.
[29]Abbo KM, Dooris M, Glazier S, et al. Features and OutCome of No-reflow After Percutaneous Coronary Intervention. Am J Cardiol, 1995; 75: 778-782.
[30]Kawaguchi R, Oshima S, Jingu M, et al. Usefulness of Virtual Histology Intravascular Ultrasound to Predict Distal Embolization for ST-Segment Elevation Myocardial Infarction. J Am Coll Cardial, 2007; 50: 1641-1646.
[31]Kawamoto T, Okura H, Koyama Y, et al. The Relationship Between Coronary Plaque Characteristics and Small Embolic Particles During Coronary Stent Implantation. J Am Coll Cardial, 2007; 50: 1635-1640.
[32]Hong YJ, Jeong MH, Choi YH, et al. Impact of Plaque Compositions on No Re-flow Phenomenon After Stent Deployment in Patients With Acute Coronary Syndrome: A Virtual Histology Intravascular Ultrasound Analysis. Eur Heart J, 2009.
[33]Nissen SE, Nicholls SJ, Wolski K, et al. Comparison of Pioglitazone vs Glimepiride o Progression of Coronary Atherosclerosis in Patients with Type 2 Diabetes: The Periscope Randomised Controlled Trial. JAMA,2008; 299: 1561-1573.
[34]Sugamura K, Sugiyama S, Matsuzawa Y et al. Benefit of Adding Pioglitazone to Successful Statin Therapy in Nondiabetic Patients with Coronary Artery Disease. Circ J, 2008; 72: 1193-1197.
[35]Ogasawara D, Shite J, Shinke T, et al. Pioglitazone Reduces the Necrotic-core Component in Coronary Plaque in Association With Enhanced Plasma Adiponectin in Patients With Type 2 Diabetes Mellitus. Circ J, 2009; 73:343-351.
[36]Nasu K, Tsuchikane E, Katoh O, et al. Effect of Fluvastatin on Progression of Coronary Atherosclerotic Plaque Evaluated by Virtual Histology Intravascular Ultrasound. JACC Cardiovasc Interv, 2009; 2: 689-696.
[37]Lee SW, Hau WK, Kong SL, et al. Virtual Histology Findings and Effects of Varying Doses of Atorvastation on Coronary Plaque Volume and Composition in Statin-Naïve Patients. The VENUS Study. Circulation J, 2012; 76: 2662-2672.
[38]Nasu K, Tsuchikane E, Katoh O, et al. Impact of Intramural Thrombus in Coronary Arteries on the Accuracy of Tissue Characterization by In-Vivo Intravascular Ultrasound Radiofrequency data Analysis. Am J Cardiol, 2008; 101: 1079-1083.