参考文献
[1]李洪珍,李铁生. 120例煤矿工人肺部病理变化与煤矽肺病理诊断的关系[J].职业卫生与病伤,1989,4(1):1-4.
[2]李铁生,李洪珍.间质硬化型矽肺的病理观察[J].中国综合临床,1980,1:30-35.
[3]李洪珍,李铁生.煤矿尘肺的病理类型[J].煤矿医学,1982,4(增刊):72-74.
[4]李洪珍,李铁生.煤矿尘肺病理形态标准[J].煤矿医学,1982,4(增刊):69-71.
[5]李洪珍,李铁生.煤矿尘肺基本的病理变化[J].煤矿医学,1982,4(增刊):65-68.
[6]李铁生,李洪珍.煤矿工人尘肺结节性病变的观察[J].煤矿医学,1981,3(2):1-4.
[7]李洪珍,李铁生.生产性粉尘在肺内沉着[J].煤矿医学,1981,3(3):31-35.
[8]李铁生,李洪珍.煤矿尘肺肺部并发症对病变的影响[J].煤矿医学,1982,4(增刊):75-77.
[9]李洪珍,王洪原,孟韶阁,等.煤矿尘肺含铁小体的形态及尘肺病变特点观察[J].中华病理杂志,1991,20(3):169-171.
[10]李洪珍,李铁生.煤尘及煤矽混合性粉尘所致大白鼠肺脏的形态学变化[J].职业卫生与病伤,1989,4(3):7-12.
[11]王献华,李铁生,杨方,等.咽泰对大鼠慢性染煤尘的实验性煤肺病变疗效的观察[J].中国煤炭工业医学杂志,1998,5(1):477-479.
[12]李洪珍,李铁生.煤矿尘肺病理组织学图谱[M].北京:能源出版社,1987.
[13]戴宇飞,秦立强,张作文. 2010—2017年职业卫生与职业病领域国家自然科学基金资助项目分析[J].中华预防医学杂志,2018,52(7):769-771.
[14]Bing Han,Hongbo Liu,Guojiang Zhai,et al.Estimates and Predictions of Coal Workers’ Pneumoconiosis Cases among Redeployed Coal Workers of the Fuxin Mining Industry Group in China:A Historical Cohort Study[J]. PLoS One,2016,11(2):e0148179.
[15]Cheng BW,Su M. International incidence trend of coal workers’ pneumoconiosis[J].中华劳动卫生职业病杂志,2019,37(1):75-78.
[16]Han S,Chen H,Harvey MA,et al. Focusing on Coal Workers’ Lung Diseases:A Comparative Analysis of China,Australia,and the United States[J]. Int J Environ Res Public Health,2018,15(11).:E2565.
[17]中国煤炭工业协会. 2017煤炭行业发展年度报告,2018-3-27.
[18]国家统计局.第三次全国经济普查,2016-3-1.
[19]李德鸿.不要把尘肺病防治引入歧途[J].环境与职业医学,2018,35(4):283-285.
[20]中华人民共和国国家卫生健康委员会.http://www.nhc.gov.cn/guihuaxxs/ s10748/201905/9b8d52727cf3 46049de8acce25ffcbd0.shtml.
[21]孟韶阁,等.矿山矽肺与结核防治[M].北京:能源出版社,1985.
[22]杨方.尘肺肺结核的病理[M]//钟明,等.尘肺合并症.北京:中国医药科技出版社,1993,45-48.
[23]Yew WW,Leung CC,Chang KC,et al. Can treatment outcomes of latent TB infection and TB in silicosis be improved?[J].J Thorac Dis,2019,11(1):E8-E10.
[24]Skowroński M1,Halicka A,Barinow-Wojewódzki A. Pulmonary tuberculosis in a male with silicosis[J].Adv Respir Med,2018,86(3). doi:10.5603.24.
[25]王光华.煤工尘肺合并肺癌31例诊治体会[J].实用医技杂志,2010,17(3):258-259.
[26]包树文.煤工尘肺合并肺癌38例分析[J].吉林医学,2012,33(18):3972.
[27]Manno M,Levy L,Johanson G,et al. silicosis and lung cancer:what level of exposure is acceptable?[J].Med Lav,2018;109(6):478-480.
[28]Sato T,Shimosato T,Klinman DM. Silicosis and lung cancer:current perspectives[J]. Lung Cancer,2018,9:91-101.
[29]Mlika M,Adigun R. Silicosis(Coal Worker Pneumoconiosis). Source stat pearls[Internet]. Treasure Island(FL):StatPearls Publishing,2019.
[30]赵培真,杨方.中国年轻人动脉粥样硬化病理生物学图谱[M].北京:中国协和医科大学出版社,2006.
[31]徐洪,杨方,袁媛,等.免疫组织化学Image Pro Plus图像半定量分析的参数选择[J].解剖学杂志,2012,35(1):37-41.
[32]杨方,赵培真,佘铭鹏,等.人冠状动脉几种细胞外基质的动态变化与动脉粥样硬化形成的关系[J].中华病理学杂志,1998,27(3):177-181.
[33]Bonan Zhang,Hong Xu,Yi Zhang,et al. Targeting the RAS axis alleviates silicotic fibrosis and AngⅡ-inducedmyofibroblast differentiation via inhibition of the hedgehog signalingpathway[J]. Toxicology Letters,2019,313:30-41.
[34]张惠,徐丁洁,毛娜,等.乙酰化微管蛋白在矽肺成纤维细胞中定位及表达变化[J].中国职业医学,2018,45(2):150-156.
[35]Deng H,Xu H,Zhang X,et al. Protective effect of Ac-SDKP on alveolar epithelial cells through inhibition of EMT via TGF-β1/ROCK1 pathway in silicosis in rat[J]. Toxicol Appl Pharmacol,2016,16(294):1-10.
[36]Li Shifeng,Xu Hong,Yi Xue,et al. Ac-SDKP increases α-TAT 1 and promotes the apoptosis in lung fibroblasts and epithelial cells double-stimulated with TGF-β1 and silica[J]. Toxicology and Applied Pharmacology,2019,369:17-29.
[37]苏敏、邹昌琪.尘肺病理诊断图谱[M].北京:人民卫生出版社,2019.
[38]邹仲之,李继承.组织学与胚胎学[J].北京:人民卫生出版社,2013.
[39]柏树令,应大君.系解剖学[J].北京:人民卫生出版社,2013.